turinabol vs anavar
One type of oral anabolic androgenic steroid (AAS) is chlorodehydromethyltestosterone. East German scientists created it in the 1960s with the goal of improving performance, especially for their Olympic athletes. Turinabol is a modified form of Dianabol (methandrostenolone) that is derived from testosterone. turinabol avis
Information in regards to Turinabol was first published in 1962 and Chlorodehydromethyltestosterone was then manufactured and released by Jenapharm in East Germany as Turinabol and Oral Turinabol. Much like other compounds such as Anavar (Oxandrolone), Turinabol was revered by medical personnel and physicians for its ability to provide a fairly distinct separation of its anabolic and androgenic effects, favoring anabolic effects of course. This is one of the reasons why its often compared to compounds such as Anavar or Promobolan. As a result, much like Anavar or Primobolan, Oral-Turinabol saw extensive medical use in not just adult males but in women and children also. At the time, Turinabol was offered in two different concentrations per tablet: 1mg tablets, and 5mg tablets. The 1mg tablets would typically be utilized for individuals traditionally more sensitive to anabolic steroid therapies, such as women and children.
Later in the 1990s, it was discovered that Turinabol was one of the key anabolic steroids utilized by East Germany in their infamous state-sponsored doping program known as the State Plan Research Theme 14.25. This plan was developed by the East German government in the late 1960s and implemented between 1974 – 1989 for the explicit purpose of administering anabolic steroids to all of their athletes (whether unbeknownst to them or not) in order to dominate at the Olympic games and other international sporting events. The core goal of this program was to simply cheat the anabolic steroid testing system in the Olympics by administering what would be at the time undetectable (due to its existence not being relatively well known) anabolic steroids to unwitting athletes, both male and female, who were simply told by their trainers and coaches that they were being given tiny blue vitamins.
Although Tbol had expressed an incredible record of valid application and safety, in 1994 Jenapharm halted production. This was a time in the early 1990s when the majority of anabolic steroids had been discontinued and pulled from markets all across the world due to the increasing anti-steroid stigma at the time. The increasing amounts of negative attention drawn to the use of anabolic steroids in sports in the early 1990s did not help Turinabol’s case, and its fate at the time was similar to many other anabolic steroids at the time as well. Jenapharm was eventually bought by Schering AG in 1996, but did not resume the manufacture of Turinabol.
CHEMICAL CHARACTERISTICS turinabol avant après
As previously mentioned, Tbol is in reality a modified form of Dianabol (Methandrostenolone), whereby it is actually a combination of the chemical structures of Dianabol and Clostebol (4-chlorotestosterone). It possesses the same general chemical structure of Dianabol along with the 4-chloro substitution that Clostebol possesses. The result is that Tbol becomes a much milder hormone than its parent hormone Dianabol. The alterations to its chemical structure remove the ability for it to be able to be aromatized into Estrogen, as well as exhibiting a far weaker androgenic strength. Turinaboltherefore possesses an anabolic rating of 54, and a very low androgenic rating of 6, making its separation between anabolic and androgenic effects very distinct and favorable.
Turinabol is C17-alpha alkylated so as to allow oral bioavailability, and as a result, will exhibit a measure of liver toxicity. It also possesses a double bond between carbon 1 and carbon 2 (also known as the 1-ene carbon), and it is this double-bond that is responsible for the reduction of androgenic strength. Lastly, as previously mentioned, a chloro group has been added at the 4th carbon, responsible for rendering it unable to aromatize as well as reducing the androgenic strength even further.
PROPERTIES OF turinabol effet
Because of its distinct separation of its androgenic to anabolic effects, it is a weaker anabolic steroid than its parent hormone Dianabol. However, the assurance with Tbol is that with any apparent muscle building capability, it will present much less in the way of androgenic effects and absolutely no estrogenic effects (due to its inability to aromatize into Estrogen). Because of its fairly weaker strength than Dianabol, the doses required to elicit effects from Tbol are considered to be quite high (this will be explained shortly in the Tbol doses section of this profile). turinabol avis
In general, athletes and bodybuilders can expect steady and quality lean mass gains with no risk of any bloating, gyno, or any other estrogenic effects. Mass and strength gains are not known to be dramatic due to its lack of anabolic strength, but steady and quality lean gains that grow consistently over time can be expected. It is also used as an ideal cutting agent during periods of fat loss or pre-contest preparation due to its inability to convert into Estrogen. Turinabol’s capabilities really shine as an adjunct to other anabolic steroids when it is run (stacked) with other anabolic steroids due to its ability to bind to SHBG. Binding to SHBG allows more of the other anabolic steroids
4-CHLORODEHYDROMETHYLTESTOSTERONE (AKA TURINABOL, ORAL TURINABOL, TBOL)
Chemical Name: 4-chloro-17a-methyl-17b-hydroxyandrosta-1,4-dien-3-one
Molecular Weight: 334.89 g/mol
Formula: C20H27ClO2
Original Manufacturer: Jenapharm
Half Life: 16 hours
Detection Time: 11 – 12 months
Anabolic Rating: 54
Androgenic Rating: 6
Turinabol is one of the anabolic steroids that is regarded as a very ‘mild’ anabolic steroid in regard to side effects. Other similar anabolic steroids in this category include Anavar (Oxandrolone) and Primobolan (Methenolone), both regarded as almost perfect anabolic steroids due to their strong dissociation between anabolic and androgenic effects, as well as the fact that they cannot convert into Estrogen at any dose at all.
ESTROGENIC SIDE EFFECTS
Turinabol is a modified form of Dianabol, and while Dianabol does hold a moderate affinity for aromatization into Estrogen, Turinabol does not at all. This is the result of its 4-chloro substitution (the chloro group that has been affixed to the 4th carbon on the steroid structure), which disallows the opportunity for the aromatase enzyme to convert Turinabol into Estrogen.
ANDROGENIC SIDE EFFECTS
Turinabol’s chemical modification of holding a double-bond between carbons 1 and 2, as well as the 4-chloro modification, grant it with a significantly reduced androgenic strength. However, this has not eliminated Turinabol’s androgenic capabilities and although it is vastly less androgenic than most other anabolic steroids, there is still a risk with androgenic side effects (especially in those very sensitive to androgenic side effects). At lower to mid-range doses, androgenic effects should rarely be experienced due to its weakness in this department, but higher doses will elicit a greater androgenic effect, especially in females. In addition, Turinabol is indeed metabolized by the 5-alpha reductase enzyme into a stronger androgenic metabolite, but the rate of 5AR reduction
The potential for androgenic Turinabol side effects include increased sebum secretion (oily skin), increased bouts of acne (linked to increased sebum secretion), bodily and facial hair growth, benign prostatic hypertrophy (BPH), and the increased risk of triggering Male Pattern Baldness (MPB) in individuals that possess the genetic trait required for the condition to manifest itself. Women will increase their chances of experiencing virilization at doses greater than 10mg per day.
HPTA AND ENDOGENOUS TESTOSTERONE PRODUCTION SIDE EFFECTS
All anabolic steroids possess the capability to suppress and/or shut down the body’s natural endogenous Testosterone production, and Turinabol side effects are no exception to this fact. Despite the fact that Turinabol exhibits perhaps the lowest androgenic rating out of all anabolic steroids, it would still nevertheless suppressive over the course of a full cycle length. It is therefore highly advised that any Turinabol user engage in a proper PCT (Post Cycle Therapy) protocol, which should always include the use of Testosterone production stimulating ancillary compounds, such as Nolvadex and/or HCG (Human Chorionic Gonadotropin) for an average PCT period of 4 – 6 weeks following the end of a cycle of any anabolic steroid regardless of how ‘mild’ it is claimed to be in terms of its impacts on the HPTA.
HEPATOTOXIC SIDE EFFECTS
Because Turinabol is in fact a C17-alpha alkylated oral anabolic steroid, it does exhibit negative effects on the liver. Although clinical data in regard to the level of liver toxicity from Turinabol is very hard to come by, logic would suggest that due to its very low androgenic strength, liver toxicity would be minor but still existent. The fact that many East German Olympic athletes prior to 1990 were administered Turinabol for several years without significant liver problems1. However, it is important to understand that in the case of the East German athletes, the dose administered was between 5 – 35mg per day, and often times at the low end of this range. Therefore, there would be a significant difference in potential hepatotoxicity between an individual running Turinabol for 8 weeks at 15mg per day,
CARDIOVASCULAR SIDE EFFECTS
Negative cardiovascular risks, side effects, and cholesterol changes are a known side effect shared by all anabolic steroids, and this side effect does apply to Turinabol side effects. Negative cardiovascular side effects resultant from anabolic steroid use involves the reduction of HDL (the good cholesterol) and increases of LDL (the bad cholesterol). The result of such changes involves an increased risk of arteriosclerosis, and the degree to which these changes occur for the worse are usually dose-dependent (with higher doses increasing the negative changes and the risks). Other factors that affect these negative cholesterol changes are: duration of use, and route of administration. turinabol avis
BENEFITS
- Tighter muscles without a puffy look
- Increase in strength
- Increase in lean muscle mass
- Ability to strongly bind to SHBG
- Increase in free testosterone
- Low-Side Effects
MEDICAL USES
Unlike most other steroids, there are no medical uses for Turinabol. As mentioned above, it was produced for the sole purpose as a performance enhancer by the East Germans.
DOSAGES
For men, I recommend 20-50mgs per day as part of a steroid stack. Many bodybuilders think they have to use it in high dosages for good results,
Women can run Tbol at tiny dosages, 2.5-7.5mgs per day max. It can cause virilization at higher dosages. Remember the East German women jokes? They are rooted in tbol use, so be very careful ladies.
Gender | Dosages |
---|---|
Male | 20-50 milligrams per day |
Female | 2.5-7.5 milligrams per day |
HALF-LIFE turinabol opiniones
The half-life is a whopping 16 hours, so once a day dosing will work fine. Though, I have seen numerous bodybuilders split the dosage into 12-hour intervals to get the “perfect” blood levels. turinabol avis
CYCLES
ORAL-ONLY: TBOL AND DBOL CYCLE
Week | Tbol | Dbol | Aromasin |
---|---|---|---|
1 | 25mgs/ED | 25mgs/ED | 12.5mgs/EOD |
2 | 25mgs/ED | 25mgs/ED | 12.5mgs/EOD |
3 | 25mgs/ED | 25mgs/ED | 12.5mgs/EOD |
4 | 25mgs/ED | 25mgs/ED | 12.5mgs/EOD |
5 | 25mgs/ED | 25mgs/ED | 12.5mgs/EOD |
6 | 25mgs/ED | 25mgs/ED | 12.5mgs/EOD |
*Use aromasin or arimidex is dose-dependent on your response to estrogenic side effects. Exemestane at around 12.5mgs/EOD should be adequate.
INJECTABLE: turinabol para que serve
Week | Tbol | Testosterone Cypionate | Aromasin |
---|---|---|---|
1 | 50mgs/ED | 500mgs/WK | 12.5mgs/ED |
2 | 50mgs/ED | 500mgs/WK | 12.5mgs/ED |
3 | 50mgs/ED | 500mgs/WK | 12.5mgs/ED |
4 | 50mgs/ED | 500mgs/WK | 12.5mgs/ED |
5 | 50mgs/ED | 500mgs/WK | 12.5mgs/ED |
6 | 50mgs/ED | 500mgs/WK | 12.5mgs/ED |
7 | 50mgs/ED | 500mgs/WK | 12.5mgs/ED |
8 | 50mgs/ED | 500mgs/WK | 12.5mgs/ED |
9 | 500mgs/WK | 12.5mgs/ED | |
10 | 500mgs/WK | 12.5mgs/ED | |
11 | 500mgs/WK | 12.5mgs/ED |
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